[1] 卫生计生委疾病预防控制局.中国居民营养与慢性病状况报告(2015 年)[M].北京:人民卫生出版社,2015.
[2] 国家心血管病中心.中国心血管病报告[M].北京:中国大百科全图书出版社,2017.
[3] Napoli C.Developmental mechanisms involved in the primary prevention of atherosclerosis and cardiovascular disease[J].Curr Atheroscler Rep,2011,13(2):170-175.
[4] Mukherjee S.Atherosclerosis: cell biology and lipoproteins-- atherosclerosis and harbingers of cardiovascular disease start in childhood and adolescence[J].Curr Opin Lipidol,2005,16(6):705-707.
[5] Sakakura K, Nakano M, Otsuka F,et al.Pathophysiology of atherosclerosis plaque progression[J].Heart Lung Circ,2013,22(6):399-411.
[6] Bastien M, Poirier P, Lemieux I,et al.Overview of epidemiology and contribution of obesity to cardiovascular disease[J].Prog Cardiovasc Dis, 2014,56(4):369-381.
[7] 王烁,董彦会,王政和,等.1985—2014年中国7~18岁学生超重与肥胖流行趋势[J].中华预防医学杂志,2017,51(4):300-305.
[8] 李双双,马传伟,席波.中国7～17 岁儿童青少年1993—2011年血压偏高变化趋势分析[J].中国学校卫生,2016,37(10):1449-1452.
[9] 侯冬青,赵小元,段佳丽,等.不同肥胖程度儿童罹患心血管代谢异常风险的横断面调查[J].中国循证儿科杂志,2014,9(2):101-106.
[10] 刘琴,董虹孛,孟玲慧,等.高血压儿童心血管结构和功能早期改变的病例对照研究[J].中华流行病学杂志,2015,36(4):332-336.
[11] Friedemann C, Heneghan C, Mahtani K, et al.Cardiovascular disease risk in healthy children and its association with body mass index: systematic review and meta-analysis[J].BMJ,2012,345:e4759.
[12] Yan Y, Hou D, Liu J, et al.Childhood body mass index and blood pressure in prediction of subclinical vascular damage in adulthood: Beijing blood pressure cohort[J].J Hypertens,2017,35(1):47-54.
[13] Liang Y, Hou D, Zhao X, et al.Childhood obesity affects adult metabolic syndrome and diabetes[J].Endocrine,2015,50(1):87-92.
[14] Liang Y, Hou D, Shan X, et al.Cardiovascular remodeling relates to elevated childhood blood pressure: Beijing Blood Pressure Cohort Study[J].Int J Cardiol, 2014,177(3):836-839.
[15] Wang L, Gao P, Zhang M, et al.Prevalence and ethnic pattern of diabetes and prediabetes in China in 2013[J].JAMA, 2017, 317(24):2515-2523.
[16] Fu JF, Liang L, Gong CX, et al.Status and trends of diabetes in Chinese children: analysis of data from 14 medical centers[J].World J Pediatr,2013,9(2):127-134.
[17] Bouret S, Levin BE, Ozanne SE.Gene - environment interactions controlling energy and glucose homeostasis and the developmental origins of obesity[J].Physiol Rev,2015,95(1):47-82.
[18] Brumana L, Arroyo A, Schwalbe NR, et al.Maternal and child health services and an integrated, life - cycle approach to the prevention of non-communicable diseases[J].BMJ Glob Health, 2017,2(3):e000295.
[19] 中国健康促进基金会骨质疏松防治中国白皮书编委会.骨质疏松症中国白皮书[J].中华健康管理学杂志, 2009,3(3):148-154.
[20] McCormack SE, Cousminer DL, Chesi A, et al.Association between linear growth and bone accrual in a diverse cohort of children and adolescents[J].JAMA Pediatr, 2017,171(9):e171769.
[21] Berger C, Goltzman D, Langsetmo L, et al.Peak bone mass from longitudinal data: implications for the prevalence, pathophysiology, and diagnosis of osteoporosis[J].J Bone Miner Res, 2010,25(9):1948-1957.
[22] Frost HM.The mechanostat: a proposed pathogenic mechanism of osteoporoses and the bone mass effects of mechanical and nonmechanical agents[J].Bone Miner, 1987,2(2):73-85.
[23] Lappe JM, Watson P, Gilsanz V, et al.The longitudinal effects of physical activity and dietary calcium on bone mass accrual across stages of pubertal development[J].J Bone Miner Res, 2015,30(1):156-164.
[24] Chen T, Liu HX, Yan HY, et al.Developmental origins of inflammatory and immune diseases[J].Mol Hum Reprod, 2016,22(8):858-865.
[25] Johnson KJ, Springer NM, Bielinsky AK, et al.Developmental origins of cancer[J].Cancer Res, 2009,69(16):6375-6377.
[26] Azad MB, Moyce BL, Guillemette L, et al.Diabetes in pregnancy and lung health in offspring: developmental origins of respiratory disease[J].Paediatr Respir Rev, 2017,21:19-26.